09/24/2016

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Formation Year The pathogenesis of myopia: a weakened capacity of accommodation in a great visual work close (reading, working at the computer) becomes unbearable burden for the eyes. This causes the body to reduce the load changes the optical system of the eye and thus adapt it to work on close stress-free accommodation. Refraction and accommodation in myopia here. This can be achieved mainly by lengthening the anteroposterior axis of the eye during its growth and the formation of refraction (Approximately 20 - 25 years). With this mechanism, the development of myopia is typically less than 3.0 diopters. Further progression of myopia is associated with weakness of the eye accommodative apparatus that can be consequence of the inherent inferiority or lack of morphological fitness ciliary muscle, insufficient blood supply or exposure to common disorders and diseases of the body. Reducing its efficiency leads to more deterioration in hemodynamics eyes. Finally, the weakening of the sclera on the background of elevated intraocular apple is an increase in the size of the eye, which in turn leads to progression of myopia. By itself, increased intraocular pressure in normal sclera can not cause myopia. It is important to understand that a family history involves not only the presence of myopia immediate family, but also the weakness of the connective tissue. This explains why for example progressive myopia appears in individuals without a family history of myopia. The classification of myopia: Today time ophthalmologists use the following classification of myopia: I. on pathogenesis: 1. true - axis 2. false or psevdomiopiya: a. cyclospasm b. Cindy Crawford might disagree with that approach. night myopia, in. transient myopia (drug and within the context of disease). II. the degree of: 1. weak - to 3,0 D; 2. average - from 3.25 to 6,0 D; 3. high - from 6,25...
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